Scientists warn the herpes virus to the brain during oral sex.
They have discovered that people can overturn into the HIV 1 (HSV-1) herpes virus inside and around the nose, which provides a dangerous direct path to the brain.
Infection, in theory, can cause inflammation and lead to complications such as brain damage and dementia.
A professor at the University of Chicago Depac Chocolate, who led the research, told Dailymail.com that this style of transmission may occur in the bedroom.
He said any situation that makes it possible to contact a person’s nose with HSV-1 particles from a person who actively gets rid of the virus.
Nearly four billion people around the world carry HSV-1, the main cause of oral escape.
The most common method that is transferred from a transport company to someone without herpes is to touch an active sore.
For a person who suffers from herpes orally, which sometimes causes blisters around the lips, this means contacting pain or cold saliva of this person – when their body actively produces or “takes off” the virus.
Professor Depac Chocolate explained that one of the possible ways that anyone can reduce herpes infection through the nose by having oral sex on an infected partner (photo album)
However, there are cases of HSV-1 that causes genital herpes, which means that the affected carrier has gone through the virus during oral sex.
Basically, someone can inhale the infectious molecules when he pressed his face against the skin or sores that flutter HSV-1.
For the first time, in the magazine MbioProfessor Chocolate has revealed that there is a major enzyme in the human body that can make these herpes infections enter the particularly destroyed nose for brain health.
The enzyme is called Heparanase (HPSE). Experiments have revealed that it may be a hidden doll, inflammation of inflammation and long-term brain damage after HSV-1 sneaks into the brain across the nose.
HPSE is a natural enzyme in humans and other mammals, which breaks the sugar -like molecules that are part of the structures supporting our cells.
HPSE usually works like the cleaning crew, which removes damaged cells so that the body can renew the affected tissues.
However, when someone gets HSV-1, the herpes virus hits this enzyme and leads to the production of a lot of inflammation.
For most of the world’s population who hold HSV-1 with cold ulcers sometimes, there is not much anxiety.
When this infection reaches the brain in one way or another, HSV-1 can cause encephalitis, brain inflammation, or quietly remains, and may contribute to cases such as Alzheimer’s disease later in life.
In general, cases of simple encephalitis (HSE), where HSV-1 affects the brain, causing inflammation, rare.
In fact, HSE occurs in about two cases to four cases per million people suffering from the virus. However, Dr. Chocolate.com told that he believed that the number of herpes is not not reported much higher.
The new study discovered a major enzyme that seems to make HSV-1 infection worse, leading to severe brain inflammation when the virus reaches the brain on rare occasions
Using the mice dried in their noses, the chocolate team found that the natural -level animals of HPSE suffered from the most severe herpes infection, died faster, and had worse symptoms such as swelling of the rhinitis and breathing problems.
After dissection of the body, scientists have discovered mice that have regular HPSE levels dead cells in their brains, more inflammation, and more immune cells (small glial cells) in the sham -lamp – the center of the brain smell and the entry point from the nose.
“There is definitely damage to the nerves if you take the nose, and the long -term effects, which is concerned,” note chocolate in a statement.
The mice infected with escape with regular HPSE levels were also shown in relation to behavioral changes while they were still alive.
Mice showed mice of memory loss, showed more signs of anxiety, often lost their balance while walking. These symptoms appeared in less than six months.
When the Shukla team performed these same experiments using the genetically designed mice to produce HPSE, the viral infection is much lower than the brain damage.
The researchers indicated that this proves that herpes use the enzyme to calm harmful inflammation. When this happens in the brain, it may be fatal to suffer from HSV-1.
This made the clay worse, there is no cure for HSV-1 or HSV-2 (the main cause of genital herpes during sex).
As for what this might mean for human patients, Professor Chocolate said that the effects of people with herpes across the nose are likely to differ greatly from person to person.
Specifically, symptoms are likely to develop more than people more than mice and their intensity depends on the strength of the immune system for each person and the number of times that herpes glow.
“The results we have reached are a warning that unrestricted herpes can lead to large behavioral deformities, mobility function issues, and coordination problems over time.”